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All games included: Hyuga no Go-Koutou, Men of War, King of the Hill, Tto Datta, The King of Fighters ’97, The King of Fighters ’99, The King of Fighters 2000: The Power of the King, Battle Arena Toshinden, World Heroes: Arcade Championship, Street Fighter III 3rd Strike, Dragon Ball Z: King of Tournament, G-Cops, .
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All Games Included:· Hyuga no Go-Koutou· Men of War· King of the Hill· Tto Datta· The King of Fighters ’97· The King of Fighters ’99· The King of Fighters 2000: The Power of the King· Battle Arena Toshinden· World Heroes: Arcade Championship· Street Fighter III 3rd Strike· Dragon Ball Z: King of Tournament· G-Cops.All Games Included:· Hyuga no Go-Koutou· Men of War· King of the Hill· Tto Datta· The King of Fighters ’97· The King of Fighters ’99· The King of Fighters 2000: The Power of the King· Battle Arena Toshinden· World Heroes: Arcade Championship· Street Fighter III 3rd Strike· Dragon Ball Z: King of Tournament· G-Cops.
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Neo Geo (186, 91) – Full – Android –
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The effect of bile acids and alcohols on the uptake of cholesterol into the Golgi complex and its transport to lysosomes and plasma membrane was examined in HepG2 cells. The uptake of labeled cholesterol was inhibited by the addition of sodium taurocholate to the medium in a dose-dependent manner, and this inhibition was readily reversible. In contrast, the transport of cholesterol from the medium to intracellular membranes was only slightly inhibited. Similar results were obtained with dehydrocholate. Neither the addition of sodium taurodeoxycholate nor the addition of sodium cholate to the medium affected either the uptake or the transport of cholesterol. Treatment of HepG2 cells with sodium taurocholate and dehydrocholate inhibited the binding of low density lipoprotein to the cell. After bile acid treatment, the rate of degradation of prelabeled low density lipoprotein was also decreased. Therefore, bile acid treatment may promote cholesterol accumulation in the Golgi complex by inhibiting the transport of cholesterol from the medium to the Golgi complex and by causing the receptor to become desensitized to the binding of ligand.Targets of p21CIP1/WAF1 induction by chemotherapeutic agents and oncogenes.
The p21CIP1/WAF1 gene is transcriptionally induced by a wide variety of chemotherapeutic agents, including alkylating agents, microtubule-targeting agents, antimetabolites, topoisomerase inhibitors, DNA-damaging agents, and antisense nucleotides. In addition to the growth-suppressive effects of p21, it can also act as an inducer of cell death, particularly following DNA damage. Furthermore, although almost all tumor cells express p21, induction of the protein does not appear to be sufficient to induce cell death of transformed cells. p21 expression also appears to be independent of a functional p53 tumor suppressor gene. Rather, p21 acts through p53-independent pathways.Vaginal infections and early pregnancy loss among women with recurrent bacterial vaginosis.
Recurrent bacterial vaginosis (RBV) is characterized by a long-lasting inflammation of the female genital tract. RBV is suspected to adversely affect reproductive outcome, but only a few studies have addressed this issue. In
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